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These conclusions provide therapeutic targets for inner ear diseases, such as targeting mitochondrial ROS, neutralizing pro-inflammatory cytokines, and appropriately increasing autophagy levels

A Novel Truncating FLAD1 Variant, Causing Multiple Acyl-CoA Dehydrogenase Deficiency (MADD) in an 8-Year-Old Boy

Subsequently, the resultant molecule undergoes oxidation through either lipoxygenase (Lox) or a non-enzymatic pathway, leading to the formation of PE-AA-OOH ( Figure 1 ) (40, 41)