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For instance, the progress of atherosclerosis involves an increased number of pro-inflammatory M1 and immunosuppressive M2 macrophages [140] as well as an enhanced accumulation of MDSCs and Tregs [141, 142]
A contributing factor to the increased sensitivity of Nrf2 / mice to inflammation may be loss of peroxisome proliferator-activated receptor (Ppar) because it is known that Nrf2 positively regulates Ppar [185] and that Ppar exerts strong anti-inflammatory effects [186]
GPX7 and GPX8 can serve as oxidative stress sensors and utilize the H 2 O 2 produced by ERO1 to facilitate protein folding [105]
Clinical Response to Tumor Necrosis Factor- Inhibitor Therapy in the Management of Cardiac Sarcoidosis
