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Overall, mounting evidence suggests that targeting CD38 and related NAD + -consuming enzymes, such as PARPs, has great potential as an NAD + -boosting target to extend human healthspan, and several pharmacological approaches are in development to inhibit CD38 and its NADase activity 232 (TABLE 1)

However, additional data was gathered with the knockout mice that indicated that while AOD9604 did not lead to significant weight loss or increase in lipolysis without the beta(3)-adrenergic receptors, there was an increase in energy expenditure and fat oxidation

Finally, one very recent study in the USA has demonstrated that in some individuals with neurological symptoms, the presence in the blood of antibodies against this receptor may interfere with the proper uptake of B12 into the brain