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Neurosciences (Riyadh)

Kiser C, Gonul CP, Olcum M, Genc S

Additionally, N-acetylcysteine administration suppressed slow and beta electroencephalographic (EEG) activities during quiet wake, further demonstrating the fatigue-inducing properties of antioxidants and the impact of redox balance on cortical circuit properties related to sleep drive

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Homocysteine Potentiates Amyloid induced Death Receptor 4 and 5mediated Cerebral Endothelial Cell Apoptosis, Blood Brain Barrier Dysfunction and Angiogenic Impairment. Aging Cell, vol
