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In the diabetic state, long-term hyperglycemia, oxidative stress and accumulation of advanced glycation end products (AGEs) cause severe damage to endothelial cells through AGE/RAGE, AMPK, NRF2 and other pathways

Traditional medications like steroids and biologics can help control symptoms but may come with difficult side effects or long-term risks

Human and rat GSTA1 share a high identity of 76%, including Cys112, which was found NAPQI-modified in our work

Contrasting iron metabolism in undifferentiated versus differentiated MO3.13 oligodendrocytes via IL-1-induced iron regulatory protein 1

doi: 10.3390/IJMS150813151, 126