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Monoclonal antibodies targeted against melanoma and ovarian tumors enhance dendritic cell-mediated cross-presentation of tumor-associated antigens and efficiently cross-prime CD8+ T cells

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A combination of prolonged oxidative damage and hepatocellular proliferation is postulated to be responsible for liver tumor development, with tumor development primarily a result of enhanced growth of preneoplastic foci

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The upregulation of IL-1 and TNF- respectively by colchicine and MCC950, however, was a bit surprising, and could possibly be explained by the interplay between these two cytokines in self-regulating each other, although further studies must be conducted to clarify this issue