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Targeting proteins for destruction by the ubiquitin system: implications for human pathobiology
In this sense, other studies have observed increased levels of TNF- in diabetic animal tissues (Satoh et al., 2003
Front Endocrinol (Lausanne) (2022) 13:900964
When hepatocyte proliferation is insufficient in individuals with chronic liver disease, alternatively activated LPCs can compensate by proliferating and differentiating into hepatocytes or cholangiocytes
This regulatory status means long-term human safety data remains limited compared to FDA-approved drugs
