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The primary pathways of disulphide-bond formation are localized in the endoplasmic reticulum (ER) of eukaryotic cells and the periplasmic space of prokaryotic cells

Inhibitors targeting these transporters, such as V-9302 and JPH203, have emerged as potential strategies for disrupting the influx of extracellular glutamine, thereby impairing its supply to intracellular metabolic pathways [61,62]

Cre-inducible Sirt3 -overexpressing mice (C57BL/6) have been described previously 61

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