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The pathophysiological processes universally converge on progressive depletion of endogenous antioxidants (e.g., SOD) and ROS-mediated amplification of macromolecular damage and cellular dysfunction [100, 555]
3C), C4 (Fig
Toxicol Appl Pharmacol 290:6673
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"Attenuation of influenza-like symptomatology and improvement of cell-mediated immunity with long-term N-acetylcysteine treatment." Eur Respir J
