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[DOI] [PMC free article] [PubMed] [Google Scholar] 237.Narwade PS, Bora VG, Mhaske PS, Sheikh AA, Biyani KR

These mechanisms are not fully understood but include central nervous system carbohydrate reduction and glycolysis inhibition, changes in neuronal excitability mediated by ATP-sensitive potassium channels through alterations in mitochondrial function, inhibition of the mammalian target of rapamycin (mTOR) pathway, and inhibition of glutamatergic excitatory synaptic transmission [72]

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