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Gut microbiota and coronary artery disease: current therapeutic perspectives
In both drug-treated and gene-knockout groups, high autophagic activity led to the death of ethanol-treated hepatocytes and mouse liver cells, while rapamycin decreased alcohol-induced lipid accumulation, and autophagy inhibition aggravated steatosis [107]
But it is known that these compounds interact with different systems modulating them, like the cholinergic nervous system (CholNS) 8 , endocannabinoid system 9 , serotoninergic nervous system, and on neurotransmitter content 10
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