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When a GPCR is exposed to its agonist over a period of many minutes to hours or days, the response is significantly reduced and is associated with decreased receptor expression at the plasma membrane, through a related process referred to as downregulation [26]
This rapid GSH loss disrupts redox homeostasis, promotes ROS accumulation, and facilitates redox-dependent protein modifications that drive apoptotic commitment
DL: Investigation, Validation, Writing review and editing
1 (Gaussian Inc, Wallingford, 2009), pp
Yap S, Naughten ER, Wilcken B, Wilcken DE, and Boers GH (2000)
