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Moreover, HDAC9 is also downregulated in the brain of AD mouse models, whereas neuronal overexpression of HDAC9 alleviates AD-related cognitive and synaptic deficits and reduces A deposition

FIGURE 3 Furthermore, MET levels in post-mortem temporal lobe samples of autistic or healthy individuals were analyzed in other studies, showing a reduction in MET transcripts and proteins in patients with ASD compared to control ones (Sugihara et al., 2007) and children with co-occurrence of gastro-intestinal diseases