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However, the exacerbation of age-related mitochondrial oxidative stress did not affect the decline of skeletal muscle respiratory function confirming that NFE2L2 is not exclusive but part of signaling pathways that are deranged during aging [106]

DOI: 10.1016/j.jantioxres.2020.05.003

If one assumes that the primary function of ferrous iron involves catalyzing the Fenton reaction to reductively cleave hydrogen peroxides and lipid hydroperoxides, initiating and propagating LPO, then the iron data clearly contradicts and is incompatible with the previous hypothesis that peroxynitrite is a critical mediator of APAP toxicity

For example, oxidized lipids like 4-HNE bind proteins, disrupting enzymes

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