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Interestingly, the mechanism by which G0S2 inhibits lipolysis is independent of ABHD5 and occurs in a non-competitive manner (52, 53)

By modulating gene expression, supporting copper-dependent enzyme functions, and promoting extracellular matrix synthesis and anti-inflammatory processes,However, its primary use remains confined to laboratory studies and cosmetic formulations rather than mainstream medical treatments

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In D-galactose-induced aging mice, oral administration of the product significantly increased NAD+ levels in liver and adipose tissue, activated mitochondrial energy metabolism pathways, improved insulin sensitivity, lowered blood glucose and lipid levels, reduced visceral fat accumulation, and increased energy expenditure, alleviating fatigue and abnormal body weight associated with aging

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