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Mechanistically, L-tryptophan upregulated the expression of GLP-1related genes (e.g., Gcg and Pcsk1 ) in intestinal L cells, further promoting GLP-1 production ( In contrast, some postbiotics may suppress GLP-1 signaling

A decrease in the level of GLP-1R contributes to neurotoxicity mediated by microglia and astrocytes, exacerbating neuron death and convulsive activity [74]

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