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Under hyperglycaemic conditions, GLP-1 inhibits glucagon secretion by -cells through paracrine mechanisms that involve -cell-derived GABA and insulin [235,236,237]

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In a retrospective analysis of more than 60,000 patients aged 40 years and older, researchers found that individuals initiating treatment with GLP-1 receptor agonists had better long-term neurological and survival outcomes than those taking other classes of antidiabetic drugs