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Quoyer, J

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5.Peralta M, Ramos M, Lipert A, Martins J, Marques A

These postnatal influences can disrupt the ovarian follicular microenvironment, leading to inflammation, oxidative stress, and hyperinsulinemia-induced androgen excess, which impair folliculogenesis and contribute to chronic anovulation and follicular arrest

It is known incorporation of G s -coupled GPCRs into endosomes and their subsequent intracellular spatial distribution and continued endosomal signaling can influence cAMP accumulation and downstream outcomes [179], [189], [23]