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doi: 10.1016/j.lfs.2020.118966 [DOI] [PubMed] [Google Scholar] 43

In mice, genetic deletion or pharmacologic inhibition of DPP4 is able to increase the homing of CXCR4+ EPC at sites of myocardial damage, resulting in a reduced cardiac remodeling and improved heart function and survival (200)

They include: Primary hallmarks, which represent the initial sources of cellular damage that accumulate over time
Subsequently, the chi-squared test with N-1 degrees of freedom and a significance level of 0.05 was conducted to determine statistical significance

A Few Things Worth Considering: Database Challenges: Rishi Desai highlights the FAERS database's limits, noting its incapacity to establish causality between drugs and side effects due to its passive and unverified data nature
