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Patients with obesity, hypertension, lipid disorders and various other risk factors have significantly increased rates of complications (Nauck et al., 2021b)
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For astrocytes, GLP-1R activation prevents conversion to neurotoxic A1 phenotypes by interrupting microglia-derived IL-1/TNF-/C1q signaling [24,47,48], thereby promoting amyloid- clearance in Alzheimer's disease models [48] and pathogenic -synuclein in PD models [47]
