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When stimulated the surface expression of platelets increases to mediate the adhesion function of activated endothelial cells, mononuclear cells and neutrophils which play a role in promoting fibrin deposition and the role in the inflammatory response and thrombosis

The potential mechanism of GLPs action in IBD may include promotion of tissue repair of injured epithelium, regulation of T cell differentiation and functions, regulation of innate immune cells such as macrophages and dendric cells, and reduction of proinflammatory cytokines (Fig

Hair loss concerns have been raised, similar to other aggressive weight loss interventions

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9d), as also reported elsewhere in GLP-1R agonists administered peripherally in mice 49