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Description
Under carefully engineered pharmacologic conditions, it may become a complementary metabolic signal that helps transform incretin therapy from appetite suppression alone into a more comprehensive strategy targeting appetite, substrate oxidation, hepatic metabolism, and energy expenditure
2022;71(Suppl 1):719
Routine check-ins monitor your progress to ensure safety and effectiveness, allowing your provider to adjust your treatment as necessary
Discrepancies were resolved by consensus, with arbitration by a third author (V.M.) when necessary
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