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Such behavior has been observed in class B GPCRs, including PTH1R and GLP-1R, which form stable -arrestin complexes and sustain both prolonged G-protein and ERK signaling from endosomes [63,64]
Other downregulated processes included those involved in the pathogenesis of a variety of cardiomyopathic states, including cardiac muscle contraction, hypertrophic cardiomyopathy, dilated cardiomyopathy, and diabetic cardiomyopathy
