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The significant reduction in total Tau levels observed in the STZ group reflects compromised microtubule stability and extensive neuronal loss rather than pathological Tau hyperphosphorylation[41]
Specifically, it has been observed to lower triglycerides and increase HDL-C in research subjects
Its synergistic triple-agonist mechanism distinguishes it from older incretin-based therapies and makes it a focus of research into advanced obesity and metabolic treatments
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