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As Drucker pointed out to me: We know that GLP-1 is not getting directly to those neurons, but it's activating pathways that turn on those neurons

Glucose stimulated insulin release was assessed in the presence of indicated concentrations of glucose and incretin receptor agonists for 30 min and the supernatant was collected, centrifuged, and processed for the ELISA as per the manufacturers protocol

Then the second-order effects started appearing

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