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This is further supported by the near-complete absence of cardiac Glp1r mRNA in Glp1r Tie2/ mice (57), suggesting that mouse cardiac Glp1r expression is primarily contributed by endothelial cells and/or immune cells

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[1] [16] Acid reflux symptoms are recognised adverse reactions in some GLP-1 product information

doi: 10.1016/j.jclinepi.2010.07.015 [DOI] [PubMed] [Google Scholar] 27
