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In particular, GLP1-RA counteract the action of angiotensin II (ANG II) inhibiting its synthesis, increasing the inactivation of its circulating form and contrasting its action on target tissue like glomerular endothelial cells and cardiomyocytes

Whilst current limited evidence does not suggest these medications directly worsen inflammatory bowel disease activity, their gastrointestinal side effectsincluding nausea, diarrhoea, and abdominal painmay overlap with Crohn's symptoms, complicating disease monitoring

Initial clinical findings suggest that GLP-1 receptor agonists could provide an effective therapeutic strategy in patients with AD and those at risk, however, there remains a lack of sufficient evidence in large clinical samples to draw any firm conclusions