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Receptor stimulation reduces ADSC production of protective cytokines, promotes reactive oxygen species (ROS) production causing oxidative damage to fibroblasts, and reduces glucose uptake by ADSCs leading to decreased ATP production and apoptosis

This contributes to reductions in body fat percentage over time

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The current evidence suggests that: Dose-dependent efficacy : Both glycaemic and weight outcomes improve with increasing doses within the therapeutic range, suggesting that very low doses are unlikely to provide substantial benefit