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Importantly, these responses could also be explained by the redundancy of insulinotropic signals from the gut or nervous system during oral versus IP glucose loads, but in the end still demonstrate that -cell GLP-1r, in and of themselves, are not necessary for oral glucose tolerance

Estimates of immunoreactivity confirmed that the muscularis contained significant immunoreactivity above background levels, whereas the mucosa was devoid of significant immunoreactivity (Fig