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Both drugs prevented MPTP-induced motor impairment, protected dopaminergic neurons in the substantia nigra, reduced the expression of the proapoptotic signaling molecule Bax, and increased the levels of the antiapoptotic molecule Bcl-2 [87]

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Long-term clinical trials demonstrated modest but statistically significant reductions in HbA1c of 0.3-0.68% when pramlintide was added to optimized insulin therapy, confirming sustained improvement in overall glycemic control