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Collectively, these results indicate that the inhibition of intestinal FXR signaling by non-12-OH FXR-antagonistic BAs is critically involved in the regulation of GLP-1 production, insulin secretion, and glucose homeostasis by T3
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- Monocytes* (MON)
Factor X binds to activated factor V (along with a calcium molecule), and this causes the activation of prothrombin into thrombin
