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The cell cycle arrest in endothelial cells exposed to tobacco smoke condensate was confirmed by the increased relative protein expression of cell cycle arrest markers P53 (Fig

Emissions from herbal cigarettes are comparable to or exceeded those from tobacco cigarettes on nearly every metric we measured

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Additionally, the study adjusted for the variable effect of the number of smoking days per week, which is important since occasional smokers smoke fewer days per week than daily smokers

To understand the underlying mechanism, we investigated the impact of colchicine on the activation of NF-B, MAPKs, and mTOR pathways in smoke-condensate-treated endothelial cells