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The reduction of NOx urinary excretion and the high concentration of nitrotyrosine in the rats plasma and organs exhibited that an interaction between nitric oxide (NO) and ROS could contribute to the pathogenesis of hypertension in BSO-treated rats (Eiserich et al., 1995)

161 Certain ALDH isoforms, such as ALDH1, ALDH3A1, and ALDH18A1, are overexpressed in tumor cells

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, designated BSPS, which elicited potent anti-inflammatory activity by decreasing pro-inflammatory mediators (TXB2, TNF-, IL-6, and IL-1) while simultaneously increasing anti-inflammatory cAMP and IL-10 levels in vivo