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however, the majority have failed clinical trials due to adverse side effects, which typically include increased blood lipid levels and/or fatty liver, suggesting that prolonged and chronic activation of hepatic GCK adversely affects hepatic lipid metabolism (69, 84)

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Collins, A

Finally, high levels of follicle-stimulating hormone (FSH) in response to estrogen deficiency might also contribute to the high BMAT and low bone mass phenotype in OVX mice as treatment with a polyclonal antibody to FSH significantly reduces BMAT volume and increases bone mass in mice (308)

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