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Furthermore, Nalm6 cells lacking FANCB were significantly sensitive to GSH inhibition even in the absence of FA, which might be a consequence of an increase in endogenous free FA, overall suggesting that GSH supply might be fundamental for Fanconi Anaemia patients (Fig
Trial registration ClinicalTrials.gov NCT03335644
Although GSH, GSSG, and GSB are exported by Mrp1, as demonstrated by the strong impairment of the respective export process by inhibition or knockout of Mrp1 (Hirrlinger et al
& Rape, M
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