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doi: 10.1007/3-211-30714-1_38

This results in impaired autophagosome-lysosome fusion, consequently activating downstream oncogenic signaling pathways such as RAS/RAF/MEK/ERK, which further drives the progression of cirrhosis-associated HCC (149)

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As a precursor of NAC, GSH participates in redox reactions that donate electrons during the detoxification of ROS as an indirect effect [66]
