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Lactate-induced MRP1 expression contributes to metabolism-based etoposide resistance in non-small cell lung cancer cells

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Critically, it is the allosteric interaction of the GPCR protomer which results in the observed cross-talk, rather than the net effect of independent neuroendocrine signalling [173]

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Both are worrisome

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HepG2-derived microsomes and EPR spectroscopy allowed us to confirm the formation of the NFT radical anion (Fig

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