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Damage to the tubular endothelial cells can increase leukocyte adhesion, platelet aggregation, and vasoconstriction, reduce blood flow to nephrons, and damage the glomerular barrier and the extensive network of peritubular capillaries (88)

The first works studying the molecular mechanisms underlying OR desensitization were reported more than 30 years ago (Gahwiler, 1981

According to the World Health Organization (WHO), validated procedures and controlled laboratory environments are important components of reproducible research workflows

The consistently low level of Glx/Cr, NAA/Cr and Lipid/Cr in R1.40 upon demyelination indicates change of brain chemistry that could be a result of diffuse axonal damage and/or metabolic shift (73, 74)