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In addition to the role of HDL in stabilizing plaques, recent studies have focused on the lesional loss of specialized proresolving mediators (SPM) versus proinflammatory factors (i.e
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Nitric oxide induces cell death by regulating anti-apoptotic BCL-2 family members
Reactive oxygen species in cancer stem cells of head and neck squamous cancer
Chemokine regulation of angiogenesis during wound healing
