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Loss of abhd5 promotes colorectal tumor development and progression by inducing aerobic glycolysis and epithelial-mesenchymal transition

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Moreover, depletion of reduced glutathione by HOSCN could also lead to an increase in oxidative stress and bacterial susceptibility to neutrophil-generated oxidants [235, 236]

The glutamate hypothesis of schizophrenia: Neuroimaging and drug development

The cells overexpressing the MG detoxification enzymes were able to detoxify the increased MG and thus, helped the cells to grow in stress conditions
