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The addition of exogenous ROS results in resensitization of cells with high VRK2 expression to GSH inhibition, which is consistent with the fact that PC cells with low expression of VRK2 are susceptible to GSH inhibition attributes to its high basal ROS level, suggesting that cells in a state of oxidative stress have enhanced dependence on the GSH system

[1] [3] This condition is typically confirmed through blood tests for anti-intrinsic factor antibodies

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These also suggest the necessity for performing large studies, analyzing multiple genes in addition to GPx-1 , and collecting detailed information on environmental and lifestyle factors to assess the true, likely complex role of GPx-1 in modifying cancer disease risk