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By mapping non-mammalian Au-binding hotspots (e.g., Grx-domain C28-C31), this work established a foundation for designing next-generation inhibitors that exploit parasite-specific structural vulnerabilities, advancing efforts to combat auranofin resistance and improve schistosomiasis therapeutics

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Raised intracellular glucose concentrations reduce aggregation and cell death caused by mutant huntingtin exon 1 by decreasing mTOR phosphorylation and inducing autophagy

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