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This narrative review consolidates both experimental and clinical data demonstrating the central role of oxidative stress in ischaemic stroke pathophysiology and explores the gut microbiotas ability to modulate these damaging processes

Furthermore, varying pH stability requirements of the redox metabolites 37,38 and the nature of whole blood as a sample, rich in proteins and heme, add to the complexity of the analyses

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Moreover, it has been shown a reduction in mTORC1 activity, combined with the enhanced synchronization of enzymes involved in glucose homeostasis

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