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10.1038/s41598-017-13739-9 60 HaghikiaA.LiX

Aberrant mPFC GABAergic synaptic transmission and fear behavior in neuroligin-2 R215H knock-in mice

In cornuammonis 1 (CA1) area of hippocampus, mitochondrial fragmentation started as early as 2 h after the reperfusion and reached the peak at 24 h

Its key mechanism lies in the activation of AMP-activated protein kinase (AMPK) , a cellular energy sensor often referred to as the bodys metabolic master switch. When AMPK is activated by MOTS-c, several critical processes occur: Improved Glucose Utilization MOTS-c enhances insulin sensitivity, enabling cells to absorb and use glucose more effectively