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These findings suggest that intranasal GHKCu has the potential to attenuate features of AD, including cognitive decline, amyloid plaque accumulation, and neuroinflammation, and provide the rationale for further studies in aging mice, for example using an AAV vector containing sequences of A42 and mutant tau [48]

However, we are unable to establish an estimate of this number or any associated burden for two reasons

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Finally, this new tissue undergoes remodelingbeing refined and strengthened over time to restore full function