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In such a situation, the ablation of GPx4 has been demonstrated to render tumor cells more susceptible to ferroptosis-eliciting agents, suggesting that GPx4 may emerge as a promising target for the development of novel therapeutic interventions (181, 182)

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Conversely, autosomal dominant mutations (classified as CMT2K) generally present a milder, later-onset phenotype, characterized by slowly progressing distal weakness and sensory loss, with maintained ambulation into adulthood [32]

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