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Gastrointestinal studies employ chemically induced lesion models (cysteamine, indomethacin, alcohol) in rats, with BPC-157 administered before or after injury

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Antonelli A, Ferrari SM, Corrado A, Di Domenicantonio A, Fallahi P

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In tight junctions, claudins regulate the interactions between occludin, tricellulin and marvelD3, which, inversely, modulate claudin oligomerization

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Modules correlated with the KO genotype were enriched for neuronal and synaptic genes involved in vesicle-mediated transport, neurotransmitter secretion, and GTPase activity, suggesting that RAMP deletion disrupts synaptic signaling networks critical for metabolic regulation [2]

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