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TMEM164 may play an important role only in certain cellular contexts because ferroptosis resistance after TMEM164 deletion has been associated with an increase in MUFA-containing ePEs, while the ferroptosis rates in cells with other types of phospholipid species were unaffected by TMEM164 deficiency, as indicated because they do not show an accumulation of MUFA-ePEs 136

[DOI] [PubMed] [Google Scholar] 85.Nicolson G.L

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A similar phenotype was observed in families with mutation of the third enzyme FAR-1, which delivers fatty alcohol for ether lipid biosynthesis [10]

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