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162,163 Paradoxically, despite enhanced cystine uptake, MCU-overexpressed PDAC exhibited typical features of ferroptosis caused by cystine deprivation
GSH deficiency hinders the expression of vitamin D-binding proteins and receptors [114,115]
To clarify this mechanism of amplification, we propose the notion of pathological transmitter-immunity cycle (Fig
RA-deficient smooth muscles are hypertrophic, hypercontractile, and pro-fibrobiotic via the increased activation of the transforming growth factor beta (TGF) signaling pathway
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