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Comprehensive toxicology evaluations reveal that the compound does not affect food intake, body temperature regulation, or basic physiological parameters at effective doses in animal models, indicating that its metabolic effects occur through specific pathway modulation rather than general physiological disruption

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The mutation of the Wilsons disease protein ATP7B has been widely used to study the direct link between hepatic high copper and development of liver pathology

Nevertheless, the role of these neurochemicals in the mechanism of early antipsychotic response remains poorly understood